2015年10月21日星期三

High Blood Pressure with PKD : Causes & Treatments

High Blood Pressure with PKD : Causes & Treatments 
Increase the activity of the sympathetic nervous system Norodrini direct vasoconstriction kidney reason, is increased vascular renal resistance, low renal blood flow, causing the units kidney brain, and promote the secretion of renin release more juxtaglomerular through renin interactions angiotensin system induce high blood pressure.

Renin-angiotensin system RAS activation can lead to sodium and high blood pressure retention. Angiotensin II allows the renal vasoconstriction, resulting in decreased renal blood flow and increased vascular resistance, and the pressure will be increased glomerular mesangial cell contraction, leading to increased permeability of protein, proteinuria, and renal glomeruli and kidney failure.

Pregnancy increases the salt sympathetic nervous system activity can increase.

Hereditary / congenital factors ethnically different pathological manifestations of renal damage from high blood pressure there are differences

Increase high blood pressure resistance state of the arteries, high blood pressure glomerular glomerular hypertension increases continued in the glomerular vascular resistance in the glomerular capillary high perfusion, filtration and high pressure across the membrane state, which affects the state of renal cell intrinsic biological and job growth. High blood pressure, high blood pressure, glomerular State is the result of kidney damage high blood pressure is a major pathophysiological mechanisms.

The kidneys caused by high blood pressure and damage is often called hardening of benign college, first affecting the glomerular arteries, the arteries and mainly Ward and arteries between the lobules. Pests have seen the end of glomerular sclerosis, severe incidence of tubular atrophy and interstitial multifocal tubular and flake, some compensatory hypertrophy, and renal interstitial fibrosis.

The kidneys caused by high blood pressure clinical manifestations of proteinuria and impaired renal function damage. Most of the patients showed microalbuminuria. Hypertensive patients with microalbuminuria bode systemic endothelial system damage, high blood pressure, poor prognosis in patients who suffer from one cardiovascular and cerebrovascular signs. It may be due to the emergence of the glomerular capillary rupture transient hematuria small number of patients. In the late tend to be twice as high blood pressure and distal tubular function of concentration, as evidenced by frequent urination at night, urine concentration may appear abnormal. High blood pressure can cause hardening of the arteries in the retina, which causes hardening of retinopathy. Hardening of the retinal arteries generally parallel with the degree of hardening of the renal arteries, the renal artery can be a large-scale case of the reaction, it is very important to fundus examination

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kidneyfailuretreat@hotmail.com Ali Aliturnt, Sosaadk.kma I am a Muslim, for you to my brother and my sister, I'll tell you at the end of the peace be upon you God's mercy and blessings be upon you

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